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Research
Study type: OtherConfidence: Very low

Myostatin Mutation Associated with Gross Muscle Hypertrophy in a Child

Schuelke M, Wagner KR, Stolz LE, et al.

Year2004
Sample sizen=1
JournalNew England Journal of Medicine
AuthorsSchuelke M, Wagner KR, Stolz LE, et al.

Evidence is still limited and needs further study

Summary

Summary

A case report of a child who was extraordinarily muscular from birth. The child carried a loss-of-function mutation (in both alleles) of MSTN, the gene encoding myostatin — a hormone that brakes muscle growth — and had far more muscle mass and greater strength than peers. It was the first demonstration that inactivating myostatin causes gross muscle hypertrophy in humans, just as in double-muscled cattle (e.g. Belgian Blue) and 'mighty mice'. This is a single case (n=1) of a rare mutation and cannot be generalized to the wider population.

Source (read the original)

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DOI: 10.1056/NEJMoa040933

Key Findings

Key findings

  • 1

    A child with a loss-of-function mutation in MSTN (myostatin) was strikingly muscular from birth

  • 2

    Muscle mass and strength far exceeded those of same-age peers

  • 3

    Inactivating myostatin causes gross muscle hypertrophy in humans, as in double-muscled cattle and mice

  • 4

    A single case report (n=1) of a rare mutation; not generalizable to the population

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